Male Pattern Baldness

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Hair loss and growth is a physiological process, subject to numerous internal and external circumstances. As a rule, hair does not separate with the root; their ends are not split or divided. These signs indicate the manifestation of alopecia.

Statistics states that hair loss manifest in 16% in under the age of 30. At 50 years, a fair degree of alopecia is discovered in 50% of the male population. Almost 80% of men become bald by the age of 70. Male pattern baldness is a sequel to age-specific endocrine changes. The secretion of the androgens, foremost testosterone control hair loss. The diagnosis of male baldness goes in the ICD-10 for L64.

What’s happening?

Initially, DHT joins the receptors of the hair bulbs but just inhibits their work. In relation to this, numerous hair defects manifest – dryness, fragility, dullness. Further, hair growth disorders begin, as they grow more slowly. In general, visually hair thins. However, short, thin and faded hairs are detected.

Then the bulbs cease to produce the shaft and a man losses hair. On average, 10-15 years after the onset of the disorder, the bulb overgrew with connective tissue. Hair growth becomes impossible, and drug stimulation or blockage of DHP are virtueless.

All these changes occur at a normality load of serum testosterone. The timing of total pattern balding varies, on average, it is about 8-20 years. Cure is efficient within the few years after the male boldness is discovered.

The rate and stage of alopecia in men is assessed on the Norwood scale. This scale reflects the process localization and total bold area.

What causes male pattern baldness?

Inherited male baldness is not life-threatening and does not coexist with secondary disorders. The key reason for male pattern baldness is inherited disposition. In 80% baldness is inherited on the maternally and only 20% on paternal.

Endocrinopathy is among triggers of baldness. Hair bulbs located in the sincipital and frontal zones are sensible to male sex hormones. Excess dehydrotestosterone often leads to the fact that hair follicles atrophy and hair fall out.

Provoking factors of pattern balding include cancer, pharmacotherapy, endocrine dysfunction, bad nutrition, stress, chronic and inflectional diseases, traumas and underperformance of hygiene rules.

It is recommended to visit a physician if hair loss starts after intake of new drugs.

The examination of a patient with alopecia:

  • Visual examination. With the help of a loupe, the physician examines the area of ​​hair loss. It is necessary to check whether there are concomitant signs of skin lesions (scaling, swelling, etc.).
  • It is needed to estimate whether the growth of hair is observed.
  • General blood test – to identify the level of erythrocytes, white blood cells, platelets and erythrocyte sedimentation rate. These indicators may deviate systemic diseases and poisonings.
  • A blood test for syphilis to feature alopecia as one of the manifestations of secondary syphilis.
  • Analysis of the main hormones – thyrotropic hormone, prolactin, androgens. Microscopy of hair. For analysis, the patient removes a number of hairs along the edge of the alopecia. The expert carefully examines the pattern.


Hairstyles. Small bald pate can be hidden with proper haircut or hairstyle. Turn to the barber for a fantastical cut.

Frizzes and crape hair cover bold head and imitate the natural look of the hair.

If male pattern baldness is a problem or it provokes tantrum, depressed state and feeling of inferiority, the doctor prescribes Minoxidil. The lotion is spread along the hair nightly. It takes several weeks to measure whether minoxidil is aimful. To retain the result and cure male pattern boldness, continue to apply the lotion. As much as 1/3 of the men receiving Minoxidil had a distinguishable change in hair growth, while another third had only a slight increase result.

Finasteride taken orally 1 mg/day inhibits the metabolism of DHT and cut its level in blood and tissues. The drug is not bound to androgen receptors and therefore does not block the effects of hormones. Finasteride is useful for the pattern balding cure only in two zones: on the vertex and forehead.

Hair transplantation. The transfer of several hair bulbs taken from the androgen-independent zones of the scalp (peripheral postcranial and parietal areas) or donor to bald androgen-dependent scalp regions has proved to be an effective tool for some patients with male baldness. Such microscopes are the best technology for many patients with male pattern baldness. The intervention has a permanent effect but can result in ingress of infection and cicatrisation.

If the genetic program is implemented due to aging, treatment is continuous. Cure lasts after hair transplantation, as it prevents transplanted hair from the premature fallout.


Preventive measures can delay the onset of the male pattern baldness. Genetically based male boldness can be delayed but not prevented. Nutrition status and regular exercises provide proper perfusion to the hair bulb. Keep emotions calm to balance homeostasis of the endocrine system and the production of hormones.

Low-level laser therapy, or LLLT, actuates low potent hair bulbs. Laser diodes provide proper blood flow, making follicles function suitably. LLLT prevents male pattern baldness from advancing. At-home laser caps are used next to other hair loss cures, including topic foams and transplantation.


  • Hogan, Daniel J., and Matthew Chamberlain. “Male pattern baldness.” Southern medical journal 93.7 (2000): 657-662.
  • Norwood, O. T. “Male pattern baldness: classification and incidence.” Southern medical journal 68.11 (1975): 1359-1365.
  • Carey, Adam H., et al. “Polycystic ovaries and premature male pattern baldness are associated with one allele of the steroid metabolism gene CYP17.” Human molecular genetics 3.10 (1994): 1873-1876.

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